In the presence of CTP, UTP becomes an allosteric inhibitor of aspartate transcarbamoylase.
| dc.creator | J R Wild | |
| dc.creator | S J Loughrey-Chen | |
| dc.creator | T S Corder | |
| dc.date.accessioned | 2019-10-17T18:38:04Z | |
| dc.date.available | 2019-10-17T18:38:04Z | |
| dc.date.issued | 1989 | |
| dc.identifier.issn | 0027-8424 | |
| dc.identifier.issn | 0744-2831 | |
| dc.identifier.issn | 1091-6490 | |
| dc.identifier.uri | https://hdl.handle.net/1969.1/185418 | |
| dc.format.extent | 1 | en |
| dc.publisher | Proceedings of the National Academy of Sciences of the United States of America | |
| dc.relation.ispartof | 1foldr Import 2019-10-08 Batch 10 | en |
| dc.title | In the presence of CTP, UTP becomes an allosteric inhibitor of aspartate transcarbamoylase. | en |
