Frs2-deficiency in cardiac progenitors disrupts a subset of FGF signals required for outflow tract morphogenesis
dc.creator | Zhang, J. | |
dc.creator | Lin, Y. | |
dc.creator | Zhang, Y. | |
dc.creator | Lan, Y. | |
dc.creator | Lin, C. | |
dc.creator | Moon, A. M. | |
dc.creator | Schwartz, R. J. | |
dc.creator | Martin, J. F. | |
dc.creator | Wang, F. | |
dc.date.accessioned | 2019-10-15T19:17:11Z | |
dc.date.available | 2019-10-15T19:17:11Z | |
dc.date.issued | 2008 | |
dc.identifier.issn | 0950-1991 | |
dc.identifier.issn | 1477-9129 | |
dc.identifier.uri | https://hdl.handle.net/1969.1/184863 | |
dc.format.extent | 11 | en |
dc.publisher | Development (Cambridge, England) | |
dc.relation.ispartof | 1foldr Import 2019-10-08 Batch 14 | en |
dc.relation.uri | 10.1242/dev.025361 | en |
dc.title | Frs2-deficiency in cardiac progenitors disrupts a subset of FGF signals required for outflow tract morphogenesis | en |