Browsing by Department "biomedical science"
Now showing items 1-8 of 8
-
(Texas A&M University, 2013-02-22)Edema, increased tissue fluid volume, may be caused by impaired lymphatic system return. This edema formation inhibits organ function. We hypothesized that increased lymphatic fluid protein concentration would cause ...
-
(Texas A&M University, 2013-02-22)Endocrine disruptive chemicals are known to produce harmful developmental effects on humans and other animals. Since substantial quantities of these chemicals are concentrated in the fat reserves of their victims, it is ...
-
(Texas A&M University, 2013-02-22)Tottering and leaner mice carry different mutations in the portion of their genetic information (tottering locus) that encodes for the α₁[A] subunit of P/Q-type voltage-gated calcium ion channels. These channels, located ...
-
(Texas A&M University, 2013-02-22)Circadian biological clocks are found in virtually all organisms and function to generate daily rhythms. The circadian clock (built by one or more oscillators) controls rhythmicity in a wide range of processes, ranging ...
-
(Texas A&M University, 2013-02-22)The growth of the Spanish-speaking population in Texas can cause barriers between many English-speaking medical personnel and Spanish-speaking patients. This research study will determine if a language barrier, considered ...
-
Pathophysiology of endotoxin: microvascular dysfunction, and the roles of VEGF and nitric oxide (NO) (Texas A&M University, 2013-02-22)Vascular endothelial growth factor (VEGF) elicits nitric oxide (NO)-dependent vasodilation and plays major roles in angiogenesis and wound healing. Although bacterial endotoxin (LPS) has been shown to alter endothelial ...
-
(Texas A&M University, 2013-02-22)Several studies have provided evidence that the administration of testosterone to vascular tissue causes vasodilation (Costarella, Yue). This study examines the role of nitric oxide (NO) as a potential mechanism of ...
-
(Texas A&M University, 2013-02-22)Tottering mice have a mutation in the calcium channel protein, alpha-1A, which decreases calcium entry into neurons through P- and Q-type high-voltage activated calcium ion channels. Because calcium is an important signaling ...