Browsing by Author "Finnell, Richard H."
Now showing items 1-8 of 8
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Hill, Denise S.; Cabrera, Robert; Wallis Schultz, Deeann; Zhu, Huiping; Lu, Wei; Finnell, Richard H.; Wlodarczyk, Bogdan J. (Behavioural Neurology, 2015)
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Zhang, Zichao; Wlodarczyk, Bogdan J.; Niederreither, Karen; Venugopalan, Shankar; Florez, Sergio; Finnell, Richard H.; Amendt, Brad A.; Shioda, Toshi (PloS One, 2011)
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Hill, Denise Suzanne (2009-05-15)Neural tube defects (NTDs) are malformations affecting about 2.6/1000 births worldwide, and 1/1000 in the United States. Their etiology remains unknown, and is likely due to interaction of genetic susceptibility factors ...
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Cho, Sunggook Gook (2012-02-14)Activation of G-protein coupled receptor 54 (GPR54) signaling generated by kisspeptins (endogenous GPR54 ligands encoded by KISS1 gene) has been known to regulate puberty and to suppress cancer metastasis. However, an ...
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Dere, Ruhee J. (Texas A&M University, 2006-08-16)Myotonic dystrophy type 2 (DM2) is caused by the extreme expansion (from < 30 repeats in normal individuals to ~ 11,000 for the full mutation in certain patients) of the repeating tetranucleotide CCTGÂCAGG sequence in the ...
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Musselman, Andrea (1993)Since teratogen-induce congenital malformations are likely to result from changes in programmed gene expression, we have monitored the level of expression of a population of 20 candidate genes in order to identify any ...
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Paschaki, Marie; Lin, Song-Chang; Wong, Rebecca Lee Yean; Finnell, Richard H.; Dollé, Pascal; Niederreither, Karen; Cooney, Austin John (PloS One, 2012)
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Hosako, Hiromi (2009-05-15)In the studies described in this dissertation, we investigated the roles of p53 in normal development, teratogen-induced apoptosis, and birth defects. In the first study, the activation of p53 and its target genes, p21, ...